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Moreover, abnormal wound healing lays a foundation for various chronic diseases, including inflammatory disorders, fibrosis, and cancer [10]

NAPQI therefore remains in its toxic form in the liver and reacts with cellular membrane (proteins and nucleic acids), resulting in widespread hepatocyte damage and death, leading to acute liver necrosis

A growing body of evidence suggests that ethanols impact on neurotrophic factors plays a crucial role in the pathophysiology of ethanol-induced depression

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Interactions of cytokines, S-adenosylmethionine, and S-adenosylhomocysteine in alcohol-induced liver disease and immune suppression