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[DOI] [PMC free article] [PubMed] [Google Scholar] 145.Odakowska M., ciskalska M., Kepinska M., Marek G., Milnerowicz H

These processes would amplify one another in positive feed-forward loops, leading to persistent impairments of the maturation and function of local parvalbumin-GABAergic neurons microcircuits and myelinated fibers of long-range macrocircuitry

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Under normal circumstances, the majority of CpG dinucleotides are methylated, but aberrations in DNA expression caused by the promoter hypermethylation of CpG islands can lead to carcinogenesis

Exposure to pro-oxidant stimuli usually induces Nrf2 activation and upregulation of antioxidant enzyme expression through binding to the antioxidant response element (ARE), localized in the antioxidant enzyme gene promoters