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The variation trend of HrGSTm1 expression level in different tissues suggests that the gene has different detoxification functions in different tissues

doi:10.1016/j.envres.2021.111028

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In estrogen biology, GSTM1 and GSTT1 serve a specific and critical function: they conjugate the reactive quinone metabolites produced when catechol estrogens particularly 4-OHE2 (4-hydroxyestradiol) are not rapidly methylated by COMT or sulfated by SULT1A1 and instead undergo further oxidation: The oxidation cascade: CYP1B1 converts estradiol to 4-OHE2 if COMT and SULT1A1 cannot clear 4-OHE2 quickly, one-electron oxidation produces 4-OHE2 semiquinone two-electron oxidation produces 4-OHE2 quinone (4-OHE2-Q)

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