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Its primary function is to disrupt the interaction between FOXO4 and the tumor suppressor protein p53, leading to the selective apoptosis of senescent cells and a deceleration of biological aging

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Specifically, IL-1 is constitutively produced and retained in keratinocytes, but in response to several stimuli, IL-1 is released, which is essentially a primary event of inflammation

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Specifically, FOXO activity is essential for the execution of apoptosis under stress conditions (Tomohiro and Sakamoto, 2008)

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