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Conventional treatments, such as Nonsteroidal Anti-Inflammatory Drugs (NSAIDs), often fail because they target inflammation, which is largely absent in chronic tendinosis

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Quantitative analysis shows that the distance from the cementoenamel junction (CEJ) to the alveolar bone crest (ABC) (CEJ-ABC) is 0.61, 0.70 and 0.66 mm at distal, median and mesial positions, respectively (Fig

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Key pathways such as c-MET and TGF-, modulated by factors like MACC1, are central to this process