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In the present study, a well established rat model of periodontitis was utilized, which involves a ligature around the cervix of the mandibular first molar tooth (9, 10)

The deficiency in autophagy upregulates p62, that binds to Keap1, thereby inhibiting the Keap1-Cul3-E3 ubiquitin ligase complex and stabilizing Nrf2 (Lau et al., 2010)

Further studies are needed to clarify whether these epigenetic modifications directly contribute to OA and how variables such as study design and tissue source may affect the observed outcomes