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In transfusion-dependent -thalassemia (-TM), iron overload and gut metabolite TMAO promote renal dysfunction by downregulating SIRT1, inducing HMGB1 release, and upregulating the ferroptosis marker ACSL4 (Zhao et al., 2023a)

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E3 ligases Arf-bp1 and Pam mediate lithium-stimulated degradation of the circadian heme receptor Rev-erb alpha

Panc-1 cells were human patient-derived pancreatic ductal adenocarcinoma (PDAC) cancer cell line (from ATCC) that carries both oncogenic KRAS and p53 mutations 33