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For instance, convincing in vitro work indicates that homocysteine may cause a specific impairment of the binding capacity of annexin II for t-PA that may alter the endothelial-dependent fibrinolytic potential (Hajjar et al, 1998)

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304 Preclinical studies established that ASO IONIS-APO(a) RX specifically targeted hepatic LPA mRNA to reduce the levels of plasma Lp(a)

We will also collect necessary data to evaluate target engagement in the CNS and disease-modifying effects in aMCI/early AD

This, in turn, impairs antioxidant defences, exacerbating the damage