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Description

Association of UCP2 866 G/A polymorphism with chronic inflammatory diseases

V., Muhonen, T

This makes GSH replacement therapy attractive, because it has the potential to interfere with the expression of the integrated HIV genome, a site that is not attacked by the currently employed antiretrovirals (AZT, ddl, ddC, D4T)

Second, the acetylation modification of the oligomycin-sensitive conferring protein increases its binding to cyclophilin D and enhances the sensitivity of the mitochondrial permeability transition pore