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6d), cariporide completely rescued neutrophil death and NET formation caused by GAPDH inhibition (Fig

PMID: 69 SternNBajicPCampbellJCapogrossoPDomesTMirandaEPet al

Although most such substances have secondary metabolic routes which become prominent following GCA depletion, the rate of metabolism is reduced enough to produce a marked accumulation of all GCA substrates in the system

Differing from Shiga toxins, the acetyltransferase toxins produced by Salmonella Enteritidis and Typhimurium inhibited translation in macrophages by acetylation of aminoacyl-tRNAs, thereby inducing Salmonella persister formation during infection 107

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