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These CD1a reactive T cells are activated by novel lipid antigens generated through exogenous PLA2, culminating in the secretion of IL-22 and IL-17A cytokines (96)

By integrating clinical data, molecular biology evidence, and innovative model systems (e.g., bone marrow organoids), this study will provide a theoretical foundation for reversing ineffective erythropoiesis

Our findings uncover an inflammatory, metabolically reprogrammed eosinophil population that promotes chronic skin inflammation by limiting protective T cell responses