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Therefore, depending on the tissue infiltrated by the B cells with EBV latency in an individual with EBV weak HLA-II alleles, the formation of EBV-infected ectopic lymphoid structures and an infection of other cell types by the increased expression of HLA-II as a result of increased IFN-, one type or another of EBV-associated disease may develop [171,172,173,174,175,176, 195]

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Patients with severe AH also have a particularly striking burden of infection

doi: 10.1111/raq.12494

Increased iHSP72 expression in hepatocytes prevents steatosis and liver injury, underscoring its role in preserving hepatic metabolic homeostasis