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Role of the human high-affinity copper transporter in copper homeostasis regulation and cisplatin sensitivity in cancer chemotherapy

This cofactor insufficiency phenotype suggests that even intact flavoproteins may lose activity when the flavin supply is compromised, expanding the scope of riboflavin-responding conditions

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Contrarily, activity of GST was reported to be reduced in fatty MASH patients compared to healthy individuals (Shearn et al

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