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Indeed, the W34A mutation, which disrupts the ability of WW domain bind to a pSer/Thr-Pro motif 44 , prevented the PIN1 interaction with CDH1-WT (Supplementary Fig

Like normal HSCs, LSCs reside in the BM niche of AML patients (23) which supports their survival and protects them against chemotherapeutic drug treatment (2426)

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Shacter E, Williams JA, Lim M, Levine RL (1994) Differential susceptibility of plasma proteins to oxidative modification: examination by western blot immunoassay