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MicroRNA-33 and the SREBP Host Genes Cooperate to Control Cholesterol Homeostasis

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And in line with our observations with exogenous GSH, the oxidation state of roGFP-iL was slightly shifted to a more reducing state in gshA , indicating a role for endogenous glutathione in the periplasmic redox homeostasis as well

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[3] If investigations reveal deficiency states, correction of the deficiency may result in resolution of the ulceration

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