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Gene and protein characterisation of the human glutathione S-transferase kappa and evidence for a peroxisomal localisation

In: HurleyWL, editor

The absence of smad4 was also found to enhance mitochondrial ATP synthase inhibitory factor (ATPIF1) activity, ATP synthase inhibition, and reduce mitochondrial membrane potential, ultimately promoting glycolysis by decreasing glucose metabolic flux and increasing glucose accumulation in the podocyte [62]

Tumor redox heterogeneity, variations in intracellular GSH levels, and discrepancies between in vitro and in vivo redox environments make precise prediction of therapeutic activation difficult

However, later on it was applied as lipid-lowering drug in hypercholesterolemic patients (Yamashita et al