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However, their contribution is context-dependent, as genetic ablation of ABCC1 does not fully prevent apoptosis-associated GSH loss in certain models 43 , indicating the existence of compensatory or alternative efflux mechanisms

In our previous report (Ghergherehchi et al., 2019a), labeling of sensory terminals in the dorsal horn following intramuscular injection was found in only 4 of 26 cases after PEG-fusion repair

Cell number was assessed by hemocytometer counting

These results provide evidence that the reduction of peripheral GSH pools increases peripheral NGF circulation that orchestrates a neuroprotective response in the CNS, at least in the striatum, through the NGF/TrkA/Akt/Nrf2 pathway

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