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Using the HT22 mouse hippocampal neurons as an in-vitro model, we found that BSO-induced glutathione depletion is associated with the oxidative activation of PDI, and the activated PDI mediates ferroptosis by catalyzing the dimerization of the inducible NOS (iNOS)

Glutathione supports both Phase I and Phase II liver detoxification, with GSH conjugation constituting a major elimination pathway for drugs, metabolic waste products, and environmental toxins

It is also worth noting that -lipoic acid, a metabolite in the cuproptosis pathway, has shown promising results in both cell-based and animal models of Wilsons disease

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doi: 10.1080/10590500500234996