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[DOI] [PubMed] [Google Scholar] Catani M, ffytche DH

Mechanistically, EGCG co-treatment led to increased apoptosis and DNA damage compared to either agent alone, as evidenced by elevated levels of cleaved caspase-3 and PARP, a decrease in antiapoptotic Bcl-2, and an increase in pro-apoptotic Bad (60)

The Journal of laboratory and clinical medicine, 133(6), 575582

CrossRef Mitochondrial bioenergetics, metabolism, and beyond in pancreatic -cells and diabetes Alejandra Mara Rivera Nieves, Brian Michael Wauford, Accalia Fu Frontiers in Molecular Biosciences.2024;[Epub] CrossRef Haplotype variability in mitochondrial rRNA predisposes to metabolic syndrome Petr Pecina, Kristna untov, Vilma Kaplanov, Guillermo Puertas-Frias, Jan ilhav, Kateina Tauchmannov, Marek Vrback, Tom ajka, Ondej Gahura, Markta Hlavkov, Viktor Strneck, Stanislav Kmoch, Michal Pravenec, Josef Houtk, Tom Mrek, A Communications Biology.2024;[Epub] CrossRef Pyrroloquinoline Quinone Alleviates Mitochondria Damage in Radiation-Induced Lung Injury in a MOTS-c-Dependent Manner Yanli Zhang, Jianfeng Huang, Shengpeng Li, Junlin Jiang, Jiaojiao Sun, Dan Chen, Qingfeng Pang, Yaxian Wu Journal of Agricultural and Food Chemistry.2024

Long-term monitoring and follow-up of patients receiving iPSC-based treatments are also necessary to evaluate their safety and efficacy [507]