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Phosphatidylethanolamine is converted to phosphatidylcholine by phosphatidylethanolamine N-methyltransferase

This lack of specificity poses a major obstacle to clinical translation, as s widespread distribution can damage healthy tissues dependent on normal iron metabolism and redox homeostasis

When pain medications are used repeatedly, many of those foundational nutrients are quietly depletedoften without obvious early symptoms

In contrast, SET7 attenuated the inhibitory effect of OTUB1 (OTU domain-containing ubiquitin aldehyde-binding protein 1) on cystine starvation/erastin-induced ferroptosis by directly interacting with non-histone OTUB1 to catalyze its methylation on lysine 122

10.1124/mol.65.6.1364 113 VercellinoI.SazanovL