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Early pharmacokinetic studies suggested that when standard, unprotected glutathione was ingested, it was rapidly cleaved into its constituent amino acids (glutamate, cysteine, and glycine) by digestive enzymes like $\gamma$-glutamyltransferase and harsh stomach acids

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in this situation, NAC reduces the risk of hepatotoxicity to 100 mg/L, the presence of renal failure or ARDS, or progression of the toxidrome despite alkalinization therapy (39)

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Raising glutathione levels in the body can decrease muscle damage, reduce recovery time, increase strength and endurance, and shift metabolism from fat production to muscle development