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A study showed that genistein could significantly reduce the methylation of lysine 9 on histone H3 and decrease both the transcriptional and translational levels of HDAC2 in a patient-derived xenograft (PDX) orthotopic mouse TNBC model (Sharma et al., 2021)

This lactate produced is released into the extracellular space and taken up by nearby neurons, where it is converted back to pyruvate and enters directly into the citric acid cycle in the mitochondria, generating ATP through oxidative phosphorylation (200, 204)