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To investigate the mechanisms involved, macrophage-specific HIF1-knockout or FXR agonist-treatment in DSS-PN mice both showed increased expression of STAT6 target genes compared to DSS-PN

The L-cystine dependent mechanism described here, in addition to findings that GLUT1 is commonly upregulated in GBM 71 , suggests that a synthetic lethal approach targeting GSH synthesis and GLUT1 activity may result in selective toxicity towards cancer cells

A number of exclusion criteria for future placebo-controlled clinical trials could be delineated: suspected pancreatic cancer

Recently, GGT1 was shown to be induced by ER stress [163]

LPS can induce massive ROS production ( Figures 5J, K ), suggesting that TCN2 knockdown prevented reactive ROS elevation and GSH depletion