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Although the association of /-Syn condensates apparently promoted LLPS of -synuclein, its corresponding nucleation and fibrillation was effectively prevented (Li et al., 2024)

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After activation, NF-B rapidly translocates to the nucleus and binds specifically to target genes, thereby initiating the transcription of Endothelin-1 (ET-1) and inflammatory cytokines, such as tumor necrosis factor (TNF), Interleukin-1 (IL-1), and Interleukin-6 (IL-6)[17]