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Thus chronic ethanol exposure may accelerate an oxidative mechanism directly and indirectly which eventually causes cell death and extensive tissue damage (Higuchi et al ., 2001

Ungurianu, A., Zanfirescu, A., & Margin, D

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For robust peer review, studies should combine at least two orthogonal redox readouts (e.g., ROS kinetics plus GSH/GSSG or lipid peroxidation), report time courses, and relate redox changes to mechanistic endpoints (e.g., death mode, pathway markers, or resistance phenotypes)

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