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Phenotypic spectrum caused by transgenic overexpression of activated Akt in the heart

3 Mechanisms underlying cell death in cardiovascular disease Cell death is a central pathological event in CVDs, contributing to the progression of conditions such as atherosclerosis, myocardial infarction, and heart failure (Figure 2) ( Figure 2 3.1 Programmed cell death in cardiovascular diseases PCD refers to tightly regulated cellular processes involving molecular pathways that orchestrate cell demise ( Apoptosis, a caspase-mediated process, is pivotal in both acute and chronic cardiovascular injury ( Autophagy is primarily a survival mechanism, but excessive autophagy contributes to cell death under stress conditions, such as hypoxia or oxidative stress ( Iron-dependent lipid peroxidation characterizes ferroptosis, which is distinct from other PCD modalities (102)

Due to the constitutive expression of this variant library, we predict that this population is likely representative of silent variants that exhibit WT-like p53 activity and are therefore dropped out of the population

Powers and Jackson, 2008)

LeeMWangCJinSWLabrecqueMPBeischlagTVBrockmanMAet alExpression of human inducible nitric oxide synthase in response to cytokines is regulated by hypoxia-inducible factor-1