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monocytogenes controlled its biphasic life cycle and transitioned from saprophytic to pathogenic by regulating the concentrations of GSH (Reniere et al., 2015)

Furthermore, a reduction in cortical parvalbumin-positive interneurons, a hallmark of SZ, can also be triggered by NMDAR antagonists 14 , suggesting that they may be particularly vulnerable to NMDAR hypofunction (the relatively young age of the rats used in the current study prevented analysis of parvalbumin-positive interneurons since they are yet to develop)

115 This finding reinforces the role of DNA methylation profiling in optimizing treatment regimens

Briefly, dried samples were resuspended in 100 l of 50 mM ammonium acetate (AA), after which excess NEM was removed using 300 l of dichloromethane