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This indicated that LA could reduce ferroptosis-mediated tubular cell death without significant dose dependence

It also stimulates the release of growth hormone through somatoliberin stimulation, connecting sleep quality directly to recovery and repair processes

As mentioned above, we explored the potential mechanisms of FA-induced system xCT suppression and found higher p53 activation in tubular cells induced by FA administration, while LA supplementation blocked the activity of p53 and abrogated the pro-ferroptosis effects

(2018a) presented in vivo results on Scly expression and activity under the absence of Sec-rich Selenop, indicating the presence of other pathways of maintaining Sec supply for Scly, which remain to be identified