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Abstract Introduction Vitamin B12 deficiency can rarely manifest as pseudo-thrombotic microangiopathy (PTMA), a hematologic syndrome that mimics thrombotic thrombocytopenic purpura (TTP)

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This results in chronic inflammation, tissue dysfunction, and age-associated diseases such as neurodegeneration, cardiovascular disorders, and cancer.3 The pro-inflammatory environment created by SASP exacerbates aging through systemic damage and diminished cellular function.33 The accumulation of senescent cells over time underscores the necessity of developing treatment strategies that selectively target these cells while preserving their beneficial roles in cancer suppression and tissue repair.34 The growing body of research on cellular senescence and its implications for aging has shifted the focus towards therapy techniques aimed at mitigating the adverse effects of senescent cells