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In autoantibody-treated mice, C-fibers had lower mechanical thresholds and higher firing frequency and total action potentials during a 0- to 100-mN force ramp stimulus, with increased firing at relatively low forces (Fig

Phosphocreatine (5-40 mM, 24 h) at different concentrations might contribute to protection of the NRK-52E cells against MGO-induced kidney injury [4]

Results indicated that although telomere length and hTERT were elevated after treatment with epitalon, actual telomerase activity did not follow the same pattern

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doi: 10.5483/BMBRep.2023-0098 Summary Keywords acute pancreatitis, mitochondrial, calcium overload, mitochondrial permeability transition pore, regulated cell death Citation Chen F, Xu K, Han Y, Ding J, Ren J, Wang Y, Ma Z and Cao F (2024) Mitochondrial dysfunction in pancreatic acinar cells: mechanisms and therapeutic strategies in acute pancreatitis