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The teams expertise was further utilized as SC, QZ, and KR focused on drafting sections related to mRNA-based therapeutic applications and managing the citation process

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The primary inflammatory mechanisms contributing to the pathogenesis and progression of EDC primarily involve dysregulated tryptophan (Trp) metabolism and dysfunction of classical neurotransmitter systems

doi: 10.1161/ATVBAHA.117.309253 171

Previously, we showed that TG2 mediates crystallin, caspase 3, and collagen crosslinking in response to oxidative stress, hypoxic stress, and DNA damage, which leads to cataract formation, apoptotic resistance, and fibrotic development, respectively 4,5,6