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Abstract Reactive oxygen species (ROS) react preferentially with certain atoms to modulate functions ranging from cell homeostasis to cell death

Therefore, interfering with ferroptosis, with the NF-B signalling pathway being a target, might be a potentially effective measure to enhance the chemosensitivity of TNBC to PTX

Hepatic tissue from ARLD patients and mice fed alcohol displayed a remarkable reduction in MAT1A localization in mitochondria, facilitated by the isomerase peptidyl-prolyl cis/ trans isomerase (PIN1) and the casein kinase (CK2)

14-ICML 35(S2 Suppl):360, 2017

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