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The PD-relevant neurotoxin salsolinol induces ferroptosis by upregulating ACSL4 expression (54)

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Specifically, EVNs target and block the infiltration of peripheral inflammatory cells via the CCR2CCL2 axis, while the released DT activates the Nrf2GPX4 pathway, inhibiting microglial ferroptosis and neuroinflammation, thereby promoting a shift toward an anti-inflammatory phenotype