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Neutral amino acid transporters (NAATs), typically the alanine serine cysteine transporter (ASCT) and the L-type amino acid transporter 2 (LAT2), mediate the cellular uptake of Cys in ordinary cells [46]

This discrepancy of iron chelation worsening neurodegeneration characterised by local iron overload raises a critical question: why does iron chelation block ferroptosis induced by pro-ferroptotic agents such as RSL3 and erastin (where there is no iron elevation in cell culture), yet fail to protect in the context of human neurodegenerative disease where iron is elevated

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