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The antiinflammatory mechanism of methotrexate depends on extracellular conversion of adenine nucleotides to adenosine by ecto-5-nucleotidase: findings in a study of ecto-5-nucleotidase gene-deficient mice

Figure 5B shows that the Cu(II)-mediated redox reaction of 3,4-diOH-TAM with 100 M NADH was observed as a decreasing absorbance at 340 nm of NADH (reduced form) for 60 min

Heterozygous knockout of p85 improved insulin sensitivity in mice and complete absence of the p85 isoform resulted in hypoglycaemia

GPx-1 prevents the accumulation of hydrogen peroxide and thereby protects against oxidative stress

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