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When excess hepcidin is transferred to the extracellular space, it inhibits the release of Fe 2+ from the cell, ultimately leading to ferroptosis

(2020) reported that fibroblasts infiltrating the TME secrete lactate to shape lipid metabolic circuits through histone acetylation, highlighting how carbon flux and acyl-donor dynamics can converge on chromatin remodeling [97]

Therefore, an increase in labile iron pool and lipid peroxidation are considered typical presentations of ferroptosis and are used as markers of ferroptotic cell death (Fig

2024 Jul 16];38(9):2132-42

buffered stress response to Maastricht Acute Stress Test