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Moreover, these therapies primarily target advanced stages of the disease and do not adequately address underlying oxidative stress and inflammation

Furthermore, we found that the response of PC to GSH-targeted treatment is largely determined by stratified expression of VRK2, which provides a theoretical basis for individualized therapy targeting metabolism

150,151 However, MAT1A-KO mice, characterized by chronic SAM deficiency, exhibit macrovesicular steatosis, mononuclear cell infiltration in periportal areas, and HCC development