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The substance has a strong affinity for macrophages expressing FPR-1 and effectively suppresses the generation of intracellular ROS and inflammatory molecules, including IL-6, TNF-, IL-1, and COX-2 (Fig

Additional studies indicate that bisphenol-A affects estrogen signaling and induces severe alterations in the proliferation and differentiation processes of NSPCs (Tiwari et al., 2016)

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