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Cotrina and Nedergaard, 2002)

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Disruption of synaptic connectivity and impaired neuronal signaling are early signs of these diseases, and this could be due to defective processing by synaptic mitochondria, which supply the ATP needed as a source of energy for these functions (Petersen et al., 2019)

Treatment of rat cortical neurons with okadaic acid reportedly inhibits PP2A activity, resulting in an abnormal increase in mitochondrial ROS and mitochondrial fission (204)

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