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Similarly, Clostridium butyricum promotes the expansion of CD4 + Foxp3 + Tregs in the intestinal lamina propria, thereby preventing colitis and attenuating hypersensitivity reactions (86)

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This dual mechanism creates a metabolic profile distinct from single-pathway agonists: insulin sensitivity improves while energy expenditure rises, a combination that neither amylin nor GLP-1 agonism alone produces