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In the IBD inflammatory microenvironment, macrophages polarize to the M1 phenotype, characterized by enhanced glycolysis, accompanied by a reduction in pyruvate dehydrogenase (PDH) activity, leading to lactate accumulation and a shift in energy supply mechanisms

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For instance, the degradation of p21, ERK3 and Cyclin G1 by the proteasome is mediated by the ubiquitination at the N-terminal methionine instead of lysine residues 12,13,14,15

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