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Dexamethasone treatment suppressed the increase in ROS levels induced by H 2 O 2 (Figures 6H, I)

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The procedures are gentle enough that even people in their 70s or beyond tolerate them well

Further investigation revealed that co-treatment with selinexor and the ferroptosis inducer RSL3 synergistically eliminates resistant cells through dual mechanisms: inhibition of nuclear export and induction of lipid peroxidation

Using the mutant GSTP-S184A and GSTP-S184D plasmids, we found that Ser184 phosphorylation is essential for GSTP to enter the nucleus and to inhibit cytoplasmic translocation of HMGB1 in LPS-activated macrophages, while the phosphorylation of GSTP at Ser42, Tyr3, Tyr7, and Tyr198 is unnecessary