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In addition, derivatives such as HTXHB have been shown to be effective in preventing mitochondrial oxidative stress and mitochondrial DNA (mtDNA) release associated with sleep deprivation, highlighting the role of these pathways in maintaining mitochondrial function [107,122]

Indeed, recent data suggest that a chronic elevation of homocysteine level might indirectly reduce methylation of DNA, RNA, proteins, and phospholipids through a concomitant increase in SAH level secondary to homocysteine-mediated reversal of the SAH hydrolase reaction (Yi et al, 2000)

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Transcriptomic, proteomic, and lipidomic approaches also identified a novel druggable target for AML that exploits the regulation of fatty acid oxidation by the mitochondrial deacetylase, Sirtuin-3 (SIRT3)

MCP-1 (CCL2), Monocyte Chemoattractant Protein-1