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BPC-157 increases angiogenesis and growth hormone receptors

BPC-157 and TB-500 operate through fundamentally different molecular mechanisms: BPC-157 modulates the nitric oxide system and upregulates growth factors including EGF, VEGF, and FGF-2 to promote cytoprotection and angiogenesis, while TB-500 sequesters G-actin monomers to regulate actin polymerization, cytoskeletal dynamics, and cell migration

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(2003) Insulin resistance in the liver-specific IGF-1 gene-deleted mouse is abrogated by deletion of the acid-labile subunit of the IGF-binding protein-3 complex: relative roles of growth hormone and IGF-1 in insulin resistance