glutathione and cellular redox control in epigenetic regulation

glutathione and cellular redox control in epigenetic regulation The role of reductase related enzymes on homoeostasis network Mitochondrial Glutathione deficiency induces epigenetic alterations

Name: glutathione and cellular redox control in epigenetic regulation The role of reductase related enzymes on homoeostasis network Mitochondrial Glutathione deficiency induces epigenetic alterations
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Glutathione deficiency induces epigenetic alterations of vitamin D metabolism genes in the livers of high fat diet fed obese mice Scientific Reports Targeting epigenetic and post translational modifications of NRF2: key regulatory factors in disease treatment Cell Death Discovery Oxidative Stress: Molecular Mechanisms, Diseases, and Therapeutic Targets PMC Glutathione responsive nanomedicine leverages tumor redox imbalance for targeted cancer theranostics Discover Oncology Springer Nature Link Redox metabolism: ROS as specific molecular regulators of cell signaling and function: Molecular Cell

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Published research compares NAC, oral glutathione, and sublingual glutathione head-to-head (Schmitt et al., 2015)

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Although these data suggest that GSH depletion per se, at least for a short period of time, is not sufficient to cause cell injury, a prolonged depletion of mucosal GSH leads to severe degeneration of intestinal mucosal epithelial cells, as shown in BSO-treated mice (Martensson et al, 1990)