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These data suggest that FGF21 deficiency induces a mitochondria-centric hypoenergy state characterized by energy incompetency, fuel inflexibility, impaired glucose utilization, and a shift toward fat and potentially ketone utilization, reducing overall energy flux capability and fast adaptation

Inhibiting EGR1-induced HMOX-1 or activating PPAR also suppresses ferroptosis in Kupffer cells [47, 83]

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In a 16-week randomized controlled trial in 92 subjects with type 2 diabetes and blood lipid abnormalities, participants took 500 mg green tea extract three times daily