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Description

471 Knocking down FTH in cardiomyocytes prevents free iron from being stored in ferritin, increases unstable iron pools, promotes intracellular ROS production, activates oxidative stress, and causes ferroptosis in cardiomyocytes, leading to age-related heart damage

consequently, adequately powered randomized trials with 24 to 52 weeks of follow-up are needed to define exposureresponse and risk profiles

Martina, J

All the plans are crossed off the calendar

Outcome measures should include not only biochemical and hormonal markers (such as E2, testosterone, SHBG, HOMA-IR, and lipid profile) but also validated clinical endpoints, including symptom severity scores, quality of life assessments, and inflammatory biomarkers