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Next, we measured serum insulin and proinsulin levels of 6-week-old Xbp1 fl/fl and Xbp1 -/- mice and found that while insulin levels (Fig

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ASU treatment reduced lipid peroxidation and neuronal apoptosis, enhanced endogenous antioxidant defenses, including superoxide dismutase and catalase, and suppressed inflammatory responses, as evidenced by decreased levels of tumor necrosis factor-alpha in the hippocampus and prefrontal cortex [61, 62]