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This conversion can be inhibited by targeting mitochondrial ROS with antioxidants or selenium, further highlighting the importance of ROS in fibroblast-to-myofibroblast conversion [81, 82]

Although Lys360 and Lys384 were reported to be probable Sirt3-dependent deacetylation sites in murine Sirt3 knockout cells 24, 26 , their acetylation mimics did not exhibit significant decreases in IDH2 activity

doi: 10.1007/s12602-019-9514-3 23 LiuYTianXHeBHoangTKTaylorCMBlanchardEet al

Overall, these data suggest that early acute inflammation is required for the correct recruitment of OPCs to the lesion site, but that chronic inflammation may impede remyelination (Figure 2)

Therefore, more experiments are needed to explore the mechanism by which KDM3B regulates ferroptosis