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Results In the 2-year study period, approximately 53,000 admissions took place in our hospital

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The mechanism of HMGB1 attenuating ASM proliferation may be related to downstream signaling involving IL-13 and TLR-4

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Mitochondrial and endoplasmic reticulum alterations in a case of amyotrophic lateral sclerosis caused by TDP-43 A382T mutation

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